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Abstract
- Pharmacological management of
arterial hypertension is based on antihypertensive
drugs with proven efficacy
on morbidity and/or mortality endpoints.
- Aliskiren is the first renin inhibitor
to reach the market.
- There are no published trials of
aliskiren with clinical endpoints. Five
double-blind short-term (8 weeks)
placebo-controlled trials showed a
moderate effect on blood pressure.
This effect was not superior to that of
other antihypertensive drugs with
which aliskiren was compared:
hydrochlorothiazide, amlodipine,
irbesartan, losartan, valsartan, lisinopril
and ramipril.
- When added to another antihypertensive
drug, aliskiren had little or no
additional effect on blood pressure. In
particular, there is no firm evidence
that adding aliskiren to amlodipine
5 mg/day is any more effective than
doubling the dose of amlodipine.
- Aliskiren has not been tested in
patients with renovascular hypertension
or severe hypertension, but pharmacological
data suggest that
aliskiren might be less effective in
these individuals.
- Overall, the adverse effect profile
of aliskiren does not seem to be any
better than that of other antihypertensive
drugs. Aliskiren contributes to
the onset of angioedema, cough, diarrhoea
and abdominal pain, hyperuricaemia,
gout attacks, kidney stones
and skin rash. Pharmacovigilance
should focus specifically on certain
adverse effects that are known to
occur with other drugs acting on the
renin-angiotensin axis, such as
angioedema, muscle disorders and
anaemia, even though few such cases
were observed with aliskiren during
clinical trials.
- Aliskiren is contraindicated during
pregnancy, as are other antihypertensive
drugs acting on the reninangiotensin
axis.